Andrea Bodnar - Publications

Publications (15)

ECB-PERS-4138

Publications By Andrea Bodnar

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Genomic signatures of exceptional longevity and negligible aging in the long-lived red sea urchin., Polinski JM, Castellano KR, Buckley KM, Bodnar AG., Cell Rep. April 1, 2024; 43 (4): 114021.
Unique age-related transcriptional signature in the nervous system of the long-lived red sea urchin Mesocentrotus franciscanus., Polinski JM, Kron N, Smith DR, Bodnar AG., Sci Rep. June 8, 2020; 10 (1): 9182.
Ocean acidification impacts spine integrity but not regenerative capacity of spines and tube feet in adult sea urchins., Emerson CE, Reinardy HC, Bates NR, Bodnar AG., R Soc Open Sci. May 1, 2017; 4 (5): 170140.
Maintenance of somatic tissue regeneration with age in short- and long-lived species of sea urchins., Bodnar AG, Coffman JA., Aging Cell. August 1, 2016; 15 (4): 778-87.
Induction of innate immune gene expression following methyl methanesulfonate-induced DNA damage in sea urchins., Reinardy HC, Chapman J, Bodnar AG., Biol Lett. February 1, 2016; 12 (2): 20151057.
Profiling DNA damage and repair capacity in sea urchin larvae and coelomocytes exposed to genotoxicants., Reinardy HC, Bodnar AG., Mutagenesis. November 1, 2015; 30 (6): 829-39.
Cellular and molecular mechanisms of negligible senescence: insight from the sea urchin., Bodnar AG., Invertebr Reprod Dev. January 30, 2015; 59 (sup1): 23-27.
Tissue regeneration and biomineralization in sea urchins: role of Notch signaling and presence of stem cell markers., Reinardy HC, Emerson CE, Manley JM, Bodnar AG., PLoS One. January 1, 2015; 10 (8): e0133860.
Comparative DNA damage and repair in echinoderm coelomocytes exposed to genotoxicants., El-Bibany AH, Bodnar AG, Reinardy HC., PLoS One. January 1, 2014; 9 (9): e107815.
Oxidative damage and cellular defense mechanisms in sea urchin models of aging., Du C, Anderson A, Lortie M, Parsons R, Bodnar A., Free Radic Biol Med. October 1, 2013; 63 254-63.
Proteomic profiles reveal age-related changes in coelomic fluid of sea urchin species with different life spans., Bodnar A., Exp Gerontol. May 1, 2013; 48 (5): 525-30.
Sea urchin coelomocytes are resistant to a variety of DNA damaging agents., Loram J, Raudonis R, Chapman J, Lortie M, Bodnar A., Aquat Toxicol. November 15, 2012; 124-125 133-8.
Age-related changes in gene expression in tissues of the sea urchin Strongylocentrotus purpuratus., Loram J, Bodnar A., Mech Ageing Dev. May 1, 2012; 133 (5): 338-47.
Marine invertebrates as models for aging research., Bodnar AG., Exp Gerontol. August 1, 2009; 44 (8): 477-84.
Lack of age-associated telomere shortening in long- and short-lived species of sea urchins., Francis N, Gregg T, Owen R, Ebert T, Bodnar A., FEBS Lett. August 21, 2006; 580 (19): 4713-7.

Genomic signatures of exceptional longevity and negligible aging in the long-lived red sea urchin., Polinski JM, Castellano KR, Buckley KM, Bodnar AG., Cell Rep. April 1, 2024; 43 (4): 114021.

Unique age-related transcriptional signature in the nervous system of the long-lived red sea urchin Mesocentrotus franciscanus., Polinski JM, Kron N, Smith DR, Bodnar AG., Sci Rep. June 8, 2020; 10 (1): 9182.

Ocean acidification impacts spine integrity but not regenerative capacity of spines and tube feet in adult sea urchins., Emerson CE, Reinardy HC, Bates NR, Bodnar AG., R Soc Open Sci. May 1, 2017; 4 (5): 170140.

Maintenance of somatic tissue regeneration with age in short- and long-lived species of sea urchins., Bodnar AG, Coffman JA., Aging Cell. August 1, 2016; 15 (4): 778-87.

Induction of innate immune gene expression following methyl methanesulfonate-induced DNA damage in sea urchins., Reinardy HC, Chapman J, Bodnar AG., Biol Lett. February 1, 2016; 12 (2): 20151057.

Profiling DNA damage and repair capacity in sea urchin larvae and coelomocytes exposed to genotoxicants., Reinardy HC, Bodnar AG., Mutagenesis. November 1, 2015; 30 (6): 829-39.

Cellular and molecular mechanisms of negligible senescence: insight from the sea urchin., Bodnar AG., Invertebr Reprod Dev. January 30, 2015; 59 (sup1): 23-27.

Tissue regeneration and biomineralization in sea urchins: role of Notch signaling and presence of stem cell markers., Reinardy HC, Emerson CE, Manley JM, Bodnar AG., PLoS One. January 1, 2015; 10 (8): e0133860.

Comparative DNA damage and repair in echinoderm coelomocytes exposed to genotoxicants., El-Bibany AH, Bodnar AG, Reinardy HC., PLoS One. January 1, 2014; 9 (9): e107815.

Oxidative damage and cellular defense mechanisms in sea urchin models of aging., Du C, Anderson A, Lortie M, Parsons R, Bodnar A., Free Radic Biol Med. October 1, 2013; 63 254-63.

Proteomic profiles reveal age-related changes in coelomic fluid of sea urchin species with different life spans., Bodnar A., Exp Gerontol. May 1, 2013; 48 (5): 525-30.

Sea urchin coelomocytes are resistant to a variety of DNA damaging agents., Loram J, Raudonis R, Chapman J, Lortie M, Bodnar A., Aquat Toxicol. November 15, 2012; 124-125 133-8.

Age-related changes in gene expression in tissues of the sea urchin Strongylocentrotus purpuratus., Loram J, Bodnar A., Mech Ageing Dev. May 1, 2012; 133 (5): 338-47.

Marine invertebrates as models for aging research., Bodnar AG., Exp Gerontol. August 1, 2009; 44 (8): 477-84.

Lack of age-associated telomere shortening in long- and short-lived species of sea urchins., Francis N, Gregg T, Owen R, Ebert T, Bodnar A., FEBS Lett. August 21, 2006; 580 (19): 4713-7.

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